Leg Ulcers

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Question Received:

  1. What is the nursing care involved for someone with chronic venous leg ulcers? Please include nursing care of safety, observations, nutrition, hygiene, wound, medications, social, discharge, and eliminations. What is the pathophysiology of venous leg ulcers?

Response:


What is the nursing care involved for someone with chronic venous leg ulcers? Please include nursing care of safety, observations, nutrition, hygiene, wound, medications, social, discharge, and eliminations. What is the pathophysiology of venous leg ulcers?

31st May 1999

Nursing care for leg ulcers

Using a team approach, the nursing care will be directed towards both the actual and potential problems that the person may have. In terms of a lower limb ulcer this will include: facilitating rest and elevation, taking a wound swab, applying a non-stick sterile dressing, administering preparations to relieve pain (if present), administering antibiotics if prescribed, and applying a four layer bandage.

The concept of four layer bandaging originated in the 1980s at Charing Cross Hospital, London. Sustained compression of over 40 mm Hg achieved with a multilayer bandage results in rapid healing of chronic venous ulcers that have failed to heal in many months of compression at lower pressures with more conventional bandages. In the four layer technique a wool bandage is applied from the base of the toes to just below the knee joint. This is then followed by the aplication of a crepe bandage. Next to this an Elset compression bandage, in a figure of eight, is applied followed by a Coban bandage.

Pathophysiology of Leg Ulcers

Congenital weakness, pregnancy, pelvic obstruction due to tumour, obesity and standing for long periods are factors which contribute towards strain being placed on both the veins and their valves. Incompetent valves in the deep and perforating veins produces a retrograde flow of blood to the superficial system which leads to venous hypertension (Haslett et al, 1999). The resulting back pressure eventually becomes transmitted to the capillary bed producing a rise in capillary hydrostatic pressure leading to oedema. Varicosed veins are dilated and tortuous veins with incompetent valves.

Varicose ulcers develop as a result of chronic venous insufficiency and increased capillary hydrostatic pressure. "Fibrinogen is forced out through the capillary walls and fibrin is deposited as a precapillary cuff blocking the diffusion of oxygen and nutrients to the skin. The skin ulcerates (usually around the medial malleolus) when a critical degree of hypoxia is reached." (Haslett et al, 1999.) Associated characteristic features are a brownish colouration of the skin due to the deposition of melanin and haemosiderin. The rupturing of blood capillaries releases red blood cells into nearby tissues, and as these cells disintegrate haemosiderin is released.

The veins of the lower limbs carry blood - generally against gravity - to the external iliac vein and thence to the common iliac vein and inferior vena cava which opens into the right atrium of the heart. Unlike arteries, the walls of veins are thinner and contain less elastic tissue and smooth muscle. To maintain unidirectional flow the large veins have a series of valves. Contraction of the nearby skeletal muscles of the lower limbs sqeezes the veins and blood is propelled upwards. The combined effects of the valves and the musculo skeletal pumps enables the blood to be pumped from the superficial veins to the deep veins.

Diagram showing arrangement of deep and superficial veins in the leg, and the action of the muscle pump

 

 

 

 

 

 

 

 

 

 

 

Diagram showing Venous Valve Action

 

 

 




References

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